Fibromyalgia

Clauw DJ. · Clinical and Translational Research at University of Michigan, Ann Arbor, MI, USA. · Nat Clin Pract Rheumatol. · Pubmed #18560385 No free full text.

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Milerad J. · No affiliation provided · Lakartidningen. · Pubmed #18478743 No free full text.

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Hartrick CT. · No affiliation provided · Pain Pract. · Pubmed #18476894 No free full text.

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Staud R, Price DD. · No affiliation provided · Pain. · Pubmed #18384959 links to  free full text

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Griffing GT. · No affiliation provided · Medscape J Med. · Pubmed #18382716 links to  free full text

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Felson DT. · No affiliation provided · Arthritis Res Ther. · Pubmed #18341701 links to  free full text

Abstract: Geographic or ethnic differences in the occurrence of disease often provide insights into causes of disease and possible opportunities for disease prevention. Persons in China appear to have a consistently lower prevalence of rheumatoid arthritis and fibromyalgia than persons in the United States and Europe; reasons for these prevalence differences might include genetic differences, differences in environmental exposures or a combination of both. With increasing obesity, gout is becoming endemic in China. Finally, symptomatic knee osteoarthritis is extremely common in China and constitutes a major public health problem there.

Weissmann G. · No affiliation provided · FASEB J. · Pubmed #18239064 links to  free full text

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Slavin KV. · No affiliation provided · Pain Med. · Pubmed #18028038 No free full text.

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Gallagher RM. · No affiliation provided · Pain Med. · Pubmed #18028037 No free full text.

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Whitehead WE. · No affiliation provided · Am J Gastroenterol. · Pubmed #17897338 No free full text.

Abstract: Twin studies have traditionally been used to assess the heritability of diseases such as irritable bowel syndrome (IBS) by comparing concordance rates in monozygotic twins (identical genetic endowment) to dizygotic twins (half of genes shared). Wojczynski et al. used twins in a novel way-they studied monozygotic twins who were discordant for IBS (but who shared identical genes) to show that the comorbidity of IBS with major depressive disorder could NOT be due to genetic influences. This paradigm provides the most rigorous method for separating genetic from environmental influences and should be adopted by other researchers. However, the authors' conclusion that major depressive disorder and IBS are part of the same pathophysiological process is questioned on the basis of (a) incomplete co-occurrence of IBS and major depressive disorder (13-45% co-occurrence) and (b) lack of specificity-the authors show that chronic widespread pain (related to fibromyalgia) and chronic fatigue are also strongly associated with IBS. This study provides precise, generalizable estimates from a large population-based study for the comorbidity of IBS with major depressive disorder, chronic widespread pain, and chronic fatigue.

Häuser W. · No affiliation provided · Schmerz. · Pubmed #17874141 No free full text.

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Allaz AF, Piguet V. · No affiliation provided · Rev Med Suisse. · Pubmed #17727164 No free full text.

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Brown RJ. · No affiliation provided · Clin Psychol Rev. · Pubmed #17707564 No free full text.

Abstract: This special issue is devoted to the topic of medically unexplained symptoms (MUS), a heterogeneous group of conditions characterized by persistent physical symptoms that cannot be explained by medical illness or injury. Although psychological factors have long been regarded as central to these problems, patients with MUS have typically been managed within medical settings and referrals to mental health services have been relatively rare. In recent years, however, interest in the psychological nature and treatment of MUS has expanded, culminating in the development of tailored psychological interventions for these conditions. This, coupled with the increasing willingness of practitioners to diagnose conditions such as chronic fatigue syndrome, fibromyalgia and irritable bowel syndrome, has led to an increase in the number of patients who are referred for psychological treatment. At present, however, many psychological therapists are unfamiliar with the literature on MUS. With this in mind, this special issue presents a series of papers that provide an overview of what is known about the nature, aetiology and treatment of medically unexplained illness. This introductory paper provides general information about the clinical presentation, diagnosis, classification, terminology and epidemiology of MUS in adults, and concludes with an examination of important areas for future development in the field. Subsequent papers address the psychological mechanisms [Deary, V., Chalder, T., & Sharpe, M. (2007-this issue). The cognitive behavioural model of medically unexplained symptoms: A theoretical and empirical review. Clinical Psychology Review; Iverson, A., Chalder, T., & Wessely, S. (2007-this issue). Gulf war illness: Lessons from medically unexplained illness. Clinical Psychology Review; Rief, W., & Broadbent, E. (2007-this issue). Explaining medically unexplained symptoms: Models and mechanisms. Clinical Psychology Review; Roelofs, K., & Spinhoven, P. (2007-this issue). Trauma and medically unexplained symptoms: Towards an integration of cognitive and neuro-biological accounts. Clinical Psychology Review] and management [Deary, V., Chalder, T., & Sharpe, M. (2007-this issue). The cognitive behavioural model of medically unexplained symptoms: A theoretical and empirical review. Clinical Psychology Review] of these conditions. A separate overview of the literature on MUS in children and adolescents is provided by Eminson [Eminson, J. (2007-this issue). Medically unexplained symptoms in children and adolescents. Clinical Psychology Review].

Huynh CN, Yanni LM, Morgan LA. · No affiliation provided · Am Fam Physician. · Pubmed #17695564 links to  free full text

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Eisinger J. · No affiliation provided · Arthritis Res Ther. · Pubmed #17626612 links to  free full text

Abstract: Autonomic nervous system dysfunction observed in fibromyalgia, characterized without exception by a sympathetic hyperactivity and hyporeactivity, has been reported. However, several studies demonstrated reduced levels of norepinephrine and neuropeptide Y at rest and after tilt table in some patients, which was improved by beta-stimulating agents. These findings support heterogeneity in fibromyalgia-associated dysautonomia. Fibromyalgia could be a generalized sympathetic dystrophy since both conditions are activated by trauma and partly linked to sympathetic mechanisms. Yet they differ on several points: hormonal and neurochemical abnormalities are observed in fibromyalgia whereas activation by peripheral trauma and hyperosteolysis are observed in reflex sympathetic dystrophy.

Yarnitsky D. · No affiliation provided · Pain. · Pubmed #17499439 No free full text.

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Harth M, Nielson WR. · No affiliation provided · J Rheumatol. · Pubmed #17477475 links to  free full text

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Gracely RH. · No affiliation provided · J Rheumatol. · Pubmed #17477474 links to  free full text

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North CS, Hong BA, Alpers DH. · No affiliation provided · World J Gastroenterol. · Pubmed #17465442 links to  free full text

Abstract: This article revisits the links between psychopathology and functional gastrointestinal disorders such as irritable bowel syndrome (IBS), discusses the rational use of antidepressants as well as non-pharmacological approaches to the management of IBS, and suggests guidelines for the treatment of IBS based on an interdisciplinary perspective from the present state of knowledge. Relevant published literature on psychiatric disorders, especially somatization disorder, in the context of IBS, and literature providing direction for management is reviewed, and new directions are provided from findings in the literature. IBS is a heterogeneous syndrome with various potential mechanisms responsible for its clinical presentations. IBS is typically complicated with psychiatric issues, unexplained symptoms, and functional syndromes in other organ systems. Most IBS patients have multiple complaints without demonstrated cause, and that these symptoms can involve systems other than the intestine, e.g. bones and joints (fibromyalgia, temporomandibular joint syndrome), heart (non-cardiac chest pain), vascular (post-menopausal syndrome), and brain (anxiety, depression). Most IBS patients do not have psychiatric illness per se, but a range of psychoform (psychological complaints in the absence of psychiatric disorder) symptoms that accompany their somatoform (physical symptoms in the absence of medical disorder) complaints. It is not correct to label IBS patients as psychiatric patients (except those more difficult patients with true somatization disorder). One mode of treatment is unlikely to be universally effective or to resolve most symptoms. The techniques of psychotherapy or cognitive-behavioral therapy can allow IBS patients to cope more readily with their illness. Specific episodes of depressive or anxiety disorders can be managed as appropriate for those conditions. Medications designed to improve anxiety or depression are not uniformly useful for psychiatric complaints in IBS, because the psychoform symptoms that sound similar to those seen in psychiatric disorders may not have the same significance in patients with IBS.

Winfield JB. · No affiliation provided · Semin Arthritis Rheum. · Pubmed #17303220 No free full text.

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Ferrari RR, Russell AS. · Department of Medicine, University of Alberta, Edmonton, Alberta, Canada. · Nat Clin Pract Rheumatol. · Pubmed #17299442 No free full text.

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Roizenblatt S. · No affiliation provided · J Bras Pneumol. · Pubmed #17278310 links to  free full text

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Hsu MC, Clauw DJ. · No affiliation provided · Arthritis Rheum. · Pubmed #17133533 links to  free full text

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Smythe H. · No affiliation provided · J Rheumatol. · Pubmed #17086600 links to  free full text

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Lorette G. · No affiliation provided · Presse Med. · Pubmed #17086125 No free full text.

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